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Researchers discover that growing up too fast may mean dying young in honey bees
sea-maid submitted, created time 1 month 3 weeks (www.biologynews.net)
Reactive oxygen species (ROS) occur as a by-product of aerobic metabolism and impair cellular function by damaging proteins, nucleotides and lipids. Organisms possess a variety of anti-oxidant mechanisms to mitigate the effects of ROS, and the oxidative stress model of aging and senescence suggests that physiological performance declines with age due to lifetime accrual of ROS-induced damage and progressively limited anti-oxidant capacity 


MedUnion submitted, created time 1 year 6 months (www.mupnet.com)
Reactive oxygen species (ROS) are currently viewed as secondary messengers that control signal transduction through the post-translational modifications of Cys-oxidation in targeting proteins. The physiological levels of ROS are generated in response to stimulation induced by extracellular ligands. Due to the labile feature of ROS, the effect of protein oxidation is normally transient. Such a character is essential for the precise control of redox-dependent signaling homeostasis 
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